Download e-book for iPad: Color atlas of pathology: pathologic principles, associated by Ursus-Nikolaus Riede, Martin Werner

By Ursus-Nikolaus Riede, Martin Werner

ISBN-10: 1588901173

ISBN-13: 9781588901170

ISBN-10: 3131277815

ISBN-13: 9783131277817

The pocket atlas is a wonderful reference paintings that provides pathologic findings in all clinically appropriate fields, utilizing top of the range pictures and concise, good dependent explanatory texts. pictures comprise colour pictures and micrographs. Electron microscopic pictures are integrated the place valuable. transparent schematic drawings illustrate strategies akin to the process infections, and so forth. textual content bins on medical and morphological points, on problems, and with easy-to-remember mnemonic summaries are trigger in colour.

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Additional info for Color atlas of pathology: pathologic principles, associated diseases, sequela

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RER cistern collapse: This is a consequence of a peroxidation injury to the membrane, such as CCl4 intoxication ( B), and is the morphologic equivalent of the onset of programmed cell death (p. 128, 132). Riede / Werner, Color Atlas of Pathology © 2004 Thieme All rights reserved. Usage subject to terms and conditions of license. A Vacuolization of the RER (liver cells, EvG) x 200 B RER cistern collapse (liver cell, TEM) x 20000 C Inclusion in the RER (TEM) x 10000 D Russell bodies (HE) x 400 E Ribosome-lamellae complexes (TEM) x 10000 F Annular lamellae (TEM) x 20000 Riede / Werner, Color Atlas of Pathology © 2004 Thieme All rights reserved.

Stabilizing disorders in which a work slowdown in refuse collection leads to cell contamination. Lysosome activity may be divided in to the following phases and functional loops: Prelysosomal Phase Heterophagy Definition: Cellular uptake of foreign particulate material (phagocytosis) and the breakdown of that material. Cellular uptake of foreign material is divided into three phases. Recognition phase: The engulfing cell or phagocyte must first recognize the foreign material as such ( A, B). This requires “detection” proteins such as immunoglobulins (p.

They function as a sort of intracellular musculature. They attach to the same membrane molecules as do the fibronectin filaments outside. Microtubular Lesions Polymerization Block Pathogenesis: Inhibited tubulin self-aggregation inhibits these processes: — Mitosis; — Discharge of secretions; — Release of lysosomes. Example: Colchicine therapy for acute gout (p. 102). Microfilament Lesions Paired Helical Filaments Definition: These lesions appear as thickened coarse filaments resembling braids ( A2) in nerve cells.

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Color atlas of pathology: pathologic principles, associated diseases, sequela by Ursus-Nikolaus Riede, Martin Werner


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